Classification of pesticide fungicides and their resistance

Pesticide fungicides are mainly used to control plant diseases. According to the mode of action, it can be divided into:

1 Protective agent: The pesticide bactericide is sprayed on the surface of the plant before the infection of the pathogen, and it plays a protective role, even if the bacteria return, it will not infect the plant. Such as Bordeaux liquid, Fumei and Daisen and organic sulfur fungicides.

2 Therapeutic agent: The pesticide bactericide is applied after the pathogen infects the plant, which can inhibit the growth and even death of the pathogen, and can alleviate the damage degree of the plant and even restore health. Pesticide fungicides with classic therapeutic effects are systemic agents, such as carbendazim, tricyclazole, propiconazole, Jinggangmycin, etc., which have strong systemic therapeutic effects. The systemic fungicides such as metalaxyl and aluminum triethylphosphinate have a two-way systemic absorption of the apical and base. It is particularly advantageous to play a therapeutic role.

3 eradication agents: pesticide fungicides directly contact plant pathogens and kill bacteria so that they can not infect plants. The scavengers have strong effects, and some can not be used in the growing season; when the concentration of the stone sulphur mixture is high, it has a scavenging effect. For example, before the germination of the peach trees, the sclerotia on the branches can be killed.

For a long time, the pests are susceptible to drug resistance. The so-called pest resistance refers to a strain of pests and diseases in the presence of a drug, forming a dose commonly used to tolerate pesticides, and this pesticide dose is still effective for most sensitive individuals in the normal population, which means that the individual has resistance Medicinal. For pesticide fungicides, the bacteria are not resistant to protective fungicides (such as sulfur, fumei series, mancozeb, Bordeaux, etc.), but to systemic fungicides (such as carbendazim, formazan, Triazolone, metalaxyl, propiconazole, antibiotics, etc.) are prone to drug resistance. This is because the inhibition of the life activity of the pathogen by the protective fungicide is a multi-site effect, and it is impossible for a pathogen to cause mutation at all sites. The effect of the systemic fungicide on the pathogen is single, and the agent only targets the single metabolic link of the pathogen, and the pathogen is easily mutated at this point of action.

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